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Sudden Cardiac Death in the Young and Athletes: Text Atlas by Gaetano Thiene, Domenico Corrado, Cristina Basso

By Gaetano Thiene, Domenico Corrado, Cristina Basso

This article atlas makes a speciality of the pathology and molecular genetics of unexpected cardiac loss of life within the younger and in athletes, proposing the state-of-the-art within the box because the foundation for improvement and implementation of better prevention techniques, together with, finally, molecular remedy that may medication the underlying organic illness. A wealth ofhigh-resolution colour photos, followed via transparent helping textual content, are provided to rfile the anatomic pathology of the cardiac ailments most often answerable for unexpected cardiac dying during this inhabitants, together with coronary artery ailments, cardiomyopathies, myocarditis, valve illnesses, conduction process abnormalities, congenital middle illnesses, and ion channel illnesses. The position of the molecular post-mortem in overcoming the constraints of morphological investigations and supplying new insights and avenues for prevention is defined. The strategy is, even though, interdisciplinary, with shut consciousness additionally to epidemiologic and scientific elements. The authors draw all through on their adventure won over 30 years in thecourse of a potential research conducted within the Veneto area, North East Italy. this article atlas should be of serious worth not just for cardiologists but additionally for geneticists, activities physicians, and citizens in cardiology and pathology."

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Additional info for Sudden Cardiac Death in the Young and Athletes: Text Atlas of Pathology and Clinical Correlates

Sample text

31). Its significance remains obscure since coronary dissection is not a typical finding in allergic syndromes and the eosinophilic infiltrate is mostly confined to the dissected coronary segment. Also the source of the blood dissecting the coronary wall is controversial. In contrast with the thoracic aorta, there are no vasa vasorum in the coronary arterial wall to potentially account for intramural hematoma. The finding of an intimal tear is quite rare at postmortem in spontaneous coronary dissection, probably because technical difficulties to detect [44] (Fig.

2 Embolism Embolism may complicate cardiac disease of the left-sided heart with mural thrombosis (atrial fibrillation, dilated cardiomyopathy, acute and chronic myocardial infarction) and the Arteritis Necrotizing noninfectious arteritis may involve the coronary arteries and account for coronary occlusion and SCD. This is the case of Kawasaki disease, which is featured by a peculiar involvement of the coronary arterial tree and characterized by a tendency to aneurysmal formation, because of tunica media inflammatory necrosis, and lumen thrombotic occlusion, leading to myocardial infarction and chronic ischemic heart disease [39–41] (Fig.

23). Because of the instantaneous death, it is impossible to predict whether these young people would have developed overt myocardial infarction, if resuscitated. Certainly, in case of fresh occlusive thrombosis, the occurrence of cardiac arrest due to ventricular fibrillation is most probably equivalent to that occurring within the first hour of myocardial infarction with out-of-the-hospital fatal outcome [25–28]. In case of transient coronary artery occlusion following vasospasm, it is also impossible to establish whether the constriction time would have been long enough to precipitate an acute myocardial infarction, either subendocardial or even transmural.

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