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Pathology Reviews • 1990 by Nelson Fausto, Janet E. Mead (auth.), Emanuel Rubin MD, Ivan

By Nelson Fausto, Janet E. Mead (auth.), Emanuel Rubin MD, Ivan Damjanov MD (eds.)

Rubin and Damjanov's most recent quantity of Pathology stories highlights the newest development within the interface of biology and affliction. vanguard ideas and experimental types that have been as soon as thought of unique or esoteric, are explored the following within the context of realizing extra absolutely the tactics inquisitive about human disease.

subject matters comprise: law of liver progress • contractile cells in lungs • mineral formation in bone • folate metabolism • the "riddle of the mast mobile" • HLA antigens • interleukin 6 • IgA nephropathy • Goodpasture syndrome • anti-basement membrane glomerulonephropathy • molecular mechanisms of oncogenesis • molecular prognosis of melanoma • inhibition of intercellular verbal exchange in carcinogenesis • DNA fix and its pathogenetic implications • human and rat mammary tumorigenesis.

Pathology reports • 1990 is vital examining for everybody keen on the mechanisms of disorder.

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Extra resources for Pathology Reviews • 1990

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I2, 198R 42. Ehrlich HI', Grislis G, Hunt TK: Evidence for the involvement of microtubules in wound contraction. Am J Surg 1:1:1:706, 1977 43. •1 Cell Physiol 116:345, 1983 44. El-Labban NG, Lee KW: Myofibroblasts in central giant cell granuloma of the jaws: an ultrastructural study. Histopathology 7:907, 19R:1 45. ')4:626, 1972 46. Evans IN, Adler KR: The lung strip: evaluation of a method to study contractility of pulmonary parenchyma. Exp Lung Res 2:1R7, 1981 35 47. 1, Low RR, Adler KB: Increased contractility of isolated lung parenchyma in an animal model of pulmonary fibrosis induced by bleomycin.

Since pulmonary fibrosis in humans is a progressive disease, the analogy to scarring and granulation tissue does not seem to follow to this end-point; as opposed to single wounds, whatever stimulus provokes a fibrotic response and myofibroblast proliferation appears to persist within the lung or, conversely, to provoke a chronic response. The literature is replete with other descriptions of proliferation of myofibroblast-like cells, usually associated with pathologic situations characterized by connective tissue retraction, hypertrophic scars, and tissue remodeling (124).

Pathology 4:139, 1972 13. Bhawan J: The myofibroblast. Am J Dermatopathol 3:73, 1981 14. Bhawan J, Bacchetta C, ,joris I, Majno G: A myofibroblastic tumor: infantile digital fibromatosis (recurrent digital fibrous tumor of childhood). Am J Pathol 94:19, 1979 15. Bissell HG, Hall HG, Parry G: How does the extracellular matrix direct gene expression? J Theor Bioi 99:31, 1982 16. Bitterman PB, Adelberg S, Crystal RG: Mechanisms of pulmonary fibrosis. Spontaneous release of tbe alveolar macropbagederived growth factors in the interstitial lung disorders.

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