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Paediatric Pathology by Colin L. Berry (auth.), Colin L. Berry MD, PhD, FRCPath

By Colin L. Berry (auth.), Colin L. Berry MD, PhD, FRCPath (eds.)

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1987). 8. 8 Spleen (g) 19 ±6 aData from Gruenwald and Minh: 1960. 9. le. > ... = ;. , ~. ~. j>. ) ------------- ------------- Sometimes a trace ------------- ------------- ------------- ------------- ------------- ------------- ------------- Sometimes a trace ------------- ------------- ------------- ------------- ------------- 3 II mo. 10 II 13-15 12-13 12-14 9-10 14-16 18-25 9 10 12-14 12-13 13-14 9-10 14-15 18-25 4-5 yr. 4-5 yr. 6-7 yr. 5-6 yr. 6-7 yr. 2~-3 yr. 7-8 yr. 12-16 yr. 3 10 4-5 yr.

Berry 45 Frequency Tota l liabil ity (genetic and environmental ) Em Fig. 4. Falconer's model. e beyond the threshold as affected and to estimate an upper limit of the heritability of the malformation from the birth frequency in the general population and the proportion of the relatives affected. With this type of model it has been calculated that heritability for pyloric stenosis is about 60%, estimated from the first-degree relatives of male patients, and 90% for female relatives of female patients.

3-4 mo. 3-4 mo. 4-5 mo. 1t-2 yr. 2}-2! yr. At birth 2t--3 yr. 8-10 yr 18 Second molar Three-fifths Three-fifths One-third Cusps united; occlusal completely calcified b Cusps united; occlusal incompletely calcified b (13-16) (14HC (l6-)c (14t-17) Central Lateral Cuspid Mand. 1st Bio. 2nd Bio. 1st Molar 2nd Molar 3rd Molar 14 16 17 15! ) 3-4 mo. 10-12 mo. 4-5 mo. It-Ii yr. 2-2} yr. At birth 2t-3 yr. 7-9 yr. -J7) Permanent Dentition Central Lateral Cuspid Mas. 1st Bio. 2nd Bio. 1st Molar 2nd Molar 3rd Molar 14 16 17 15!

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