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Congenital Heart Diseases: An Updated Approach to Some by Raúl, M.D., Ph.D. Cayré, José, M.D., Ph.D. Milei

By Raúl, M.D., Ph.D. Cayré, José, M.D., Ph.D. Milei

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Extra info for Congenital Heart Diseases: An Updated Approach to Some Important Issues

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Fyfe, A; Perloff, JK; Niwa, K; Child, JS; Miner, PD. Cyanotic congenital heart disease and coronary artery atherogenesis. Am J Cardiol 2005, 96, 283-90. ; Richardson, M; Rosenfeld, ME; Schaffer, SA; Schwartz, CJ; Wagner, WD; et al. A definition of the intima of human arteries and of its atherosclerosis-prone regions. A report from the Committee on Vascular Lesions of the Council on Arteriosclerosis, American Heart Association. Arterioscler Thromb 1992, 12, 120-34. Stehbens, WE. The pathogenesis of arterial aneurysms and associated lesions.

The relationship between the frequency of intimal hyperplasia and the expression of TGF-β1 and ERα in arteries from 98 pediatric patients with congenital heart defects indicated that: 1) intimal hyperplasia was a common finding in the coronary tree of these patients, 2) both TGF-β1 and ERα seemed to play a major role in this phenomenon and 3) surgical correction of CHD was associated with further coronary vascular remodeling. Introduction Congenital heart disease (CHD) patients represent a risk group for premature atherosclerotic coronary artery disease [1].

Molecular mechanisms in intimal hyperplasia. J Pathol 2000, 190, 300-9. [48] Khan, R; Agrotis, A; Bobik, A. Understanding the role of transforming growth factorbeta1 in intimal thickening after vascular injury. Cardiovasc Res 2007, 74, 223-34. [49] Kanzaki, T; Tamura, K; Takahashi, K; Saito, Y; Akikusa, B; Oohashi, H; Kasayuki, N; Ueda, M; Morisaki, N. In vivo effect of TGF-β1. Enhanced intimal thickening by administration of TGF-β1 in rabbit arteries injured with a balloon catheter. Arterioscler Thromb Vasc Biol 1995, 15, 1951-7.

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