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Diagnostic Imaging

Cerebral Visual Disturbance in Infantile Encephalopathy by O. Nieuwenhuizen

By O. Nieuwenhuizen

The analysis and therapy of kids being affected by childish encephalopathy is a part of day-by-day childneurological perform. during the last years, a large amount of wisdom has been accumul­ ated on motor and psychological handicaps, orthopaedic corrections, rehabilitation, measurements, and so forth. much less cognizance has, even though, been paid to the perceptual deficits in teenagers struggling with childish encephalopathy, e.g. visible and listening to loss. it truly is most likely that such deficits can have a unfavourable impression on improvement. As those teenagers are frequently additionally mentally retarded, perceptual deficits might be tricky to diagnose. increasing the to be had wisdom on those deficits may well facilitate their (early) prognosis. evaluate of analysis allows adaption of developmental stimulation which can final give a contribution to a better developmental point. This learn offers with the visible difficulties of youngsters struggling with childish encephalopathy. specifically, a learn has been made from visible impairment because of disorder of the relevant visible procedure (from optic chiasm to striate cortex). The childneurological features of this handicap have been investigated intimately in coopera­ tion with Prof. Dr. J. Willemse (Department of Childneurology, college health facility Utrecht, The Netherlands), leading to an identifiable scientific photo. the applying of neuro-imaging options (computer tomography, magazine­ netic resonance imaging) allows the overview of attribute morphological abnormalities of the vital visible process.

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Extra info for Cerebral Visual Disturbance in Infantile Encephalopathy

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Other regions of importance for the production of saccades are: the parietal lobe (more from the point of view of visual attention than motoric tasks), the cerebellum, the thalamus and the basal ganglia (see below). g. because of eye muscle disorders or eye muscle-nerve pareses). g. by cerebellar disorders). g due to oculomotor apraxia and Parkinson's disease). e. interfere with foveal fixation (because of cerebellar disorders). g. in brain stem disorders) (Leigh and Zee 1983c). The anatomical structures involved in the production of the slow phase have not yet been completely elucidated.

G. FPL) are easily applicable in multi-handicapped children. When this study was started, however, the FPLtechnique was not yet available in the test centre. Visual acuity was, therefore, measured by the STYCAR and other matching tests (see Chapters II and VII). Mohn and Van Hof-van Duin (1983) reported on the detection of visual field defects in children with neurological disorders. They used STYCAR balls mounted on thin black sticks to assess visual field size. When the child fixated a ball in front of him/her, a second ball was slowly moved from the periphery to the fixation point.

Amplification. The electrodes were connected with a Devices Limited High Gain Pre-Amplifier 3542. Calibration signal amplitude was 10microV. 3 s. 1 s. in the case of much noise) were chosen. Averaging and Registration. Flash-VEPs: A Datalab Signal Averager DL 102 was used. Sweep time was SOOms; each record consisted of 100 sample points. The records were plotted at a speed of 50 ms/sample point using a Bryans 25000 XY recorder. Pattern-VEPs: the signal was averaged and registered on a TV screen by the Apple device.

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